By Ramana S. Moorthy MD
Starts off with an in-depth evaluation of immunemedicated eye sickness, summarizing easy immunologic techniques, ocular immune responses and designated issues in ocular immunology. Discusses the medical method of uveitis and experiences noninfectious (autoimmune) and infectious varieties of uveitis, with an multiplied part on viral uveitis and new fabric on infectious and noninfectious scleritis. more suitable detection of infectious brokers via immunologic and genetic equipment and new biologic therapeutics are designated. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular features of AIDS. encompasses a variety of new colour photos. significant revision 2011-2012
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Additional info for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)
In particular, the Fab region on each molecule contains the antigen recognition -combining domain, called the hypervariable region. The opposite end of the molecule, on the heavy chain portion, contains the attach ment site for effector cells (the Fe portion); it also contains the site of other effector functions, such as complement activation (for IgG3) or binding to the secretory component so it can be transported through epithelia and secreted into tears (for IgA). Table 2-2 summarizes the important structural differences among immunoglobulin isotypes.
HLA-A. -8. and -C) serve as the antigen-presenting platform for CD8 T lymphocytes (Fig 2-2). CD8 T lymphocytes include natural killer T cells and regulatory T cells. Class I molecules are present on almost all nucleated cells. In general. class I APCs are best for processing peptide antigens that have been synthesized by the host cell itself. including most tumor peptides or viral peptides after host cell invasion. MHC class II molecules (ie. HLA-DR. -DP. and -DQ) serve as the antigen-presenting platform for CD4.
2nd ed. Philadelphia, PA: WB Saunders; 2000:74-82. Lymphocyte-Mediated Effector Responses Oelayed hypersensitivity T lymphocytes Delayed hypersensitivity (Coombs and Gell type IV) represents the prototypical adaptive immune mechanism for lymphocyte-triggered inflammation. It is especially powerful in secondary immune responses. Previously primed DH CD4 T lymphocytes leave the lymph node, home into local tissues where antigen persists, and become activated by further restimulation with the specific priming antigen and MHC class II - expressing CLINICAL EXAMPLE 2·2 Identification of rubella virus reactivity in Fuchs heterochromic iridocyclitis Fuchs heterochromic iridocyclitis (FHI) is a unilateral chronic anterior uveitis that frequently features elevated intraocular pressure, early cata ract, and iris atrophy leading to heterochromia.