2007-2008 Basic and Clinical Science Course Section 4: by American Academy of Ophthalmology, Debra J. Shetlar, MD

By American Academy of Ophthalmology, Debra J. Shetlar, MD

Discusses advances within the analysis and class of tumors because it courses the reader via a logical, tissue-specific series that levels from topography via affliction method to normal and differential analysis. comprises many new colour pathologic and scientific pictures and diagrams. Covers wound fix; specimen dealing with, together with processing and marking; and diagnostic suggestions. additionally encompasses a list for soliciting for ophthalmic pathologic session.

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Additional info for 2007-2008 Basic and Clinical Science Course Section 4: Ophthalmic Pathology and Intraocular Tumors

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Nat Med 2001; 7(11):1194-1201. 56. Matsumoto T, Claesson-Welsh L. VEGF receptor signal transduction. Sci STKE 2001; 2001(112):RE21. 57. Gille H, Kowalski J, Yu L et al. A repressor sequence in the juxtamembrane domain of Flt-1 (VEGFR-1) constitutively inhibits vascular endothelial growth factor-dependent phosphatidylinositol 3'-kinase activation and endothelial cell migration. EMBO J 2000; 19(15):4064-4073. 58. Chou MT, Wang J, Fujita DJ. Src kinase becomes preferentially associated with the VEGFR, KDR/Flk-1, following VEGF stimulation of vascular endothelial cells.

This was based on the knowledge that VEGF had already been shown to promote the growth of blood vessels,^ and on the observation that VEGF mRNA dramatically increases under hypoxic conditions in various cell lines. In addition, it was found that VEGF levels were increased in the hypoxic centre of tumours, suggesting that this factor could mediate the growth of new vessels into tumours. This had important clinical implications, because it was known since the early seventies that tumour growth requires sprouting of new vessels from preexisting vessels, a process known as angiogenesis.

Migration of human monocytes in response to vascular endothelial growth factor (VEGF) is mediated via the VEGF receptor flt-1. Blood 1996; 87(8):3336-3343. 36. Dikov MM, Ohm JE, Ray N et al. Differential roles of vascular endothelial growth factor receptors 1 and 2 in dendritic cell differentiation. J Immunol 2005; 174(l):215-222. 37. Nomura M, Yamagishi S, Harada S et al. Possible participation of autocrine and paracrine vascular endothelial growth factors in hypoxia-induced proliferation of endothelial cells and pericytes.

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